Events and speakers

Five things you wanted to know about epigenetics (But were afraid to ask)

10960203_781430608590258_4910408420147962125_oWhat the heck is it, anyway?

Epigenetics involves changes in how your genes work.

In classical genetics, traits pass from generation to generation in DNA, the strands of genetic material that encode your genes. Scientists thought alterations to the DNA itself was the only way changes could pass on to subsequent generations.

So say you lost a thumb to a angry snapping turtle: Because your DNA hasn’t changed, your children won’t be born with smaller thumbs. Classic.

Things get way more complicated with epigenetics. It turns out that some inherited changes pass on even though they are not caused by direct changes to your DNA. When cells divide, epigenetic changes can show up in the new cells.

Getting nibbled on by an irate turtle isn’t likely to epigenetic changes, but other factors such as exposure to chemicals and an unhealthy diet, could cause generation-spanning epigenetic changes.


How does it work?

The main players in epigenetics are histones and methyl groups.

Imagine your genes are like pages in a really long book. Prior to the mid-1800s, books came with uncut edges, so in order to read the book, you’d have to slice apart the uncut pages. That’s sort of what a histone does to DNA: They are proteins that wrap DNA around themselves like thread on a spool. They keep the DNA organized and help regulate genes.

Methyl groups (variations on CH3) attach to the histones and tell them what to do. These molecules are like notes in a book’s margin that say, “These next few pages are boring, so don’t bother cutting them open.” As you read the book, you’ll save time and effort by skipping some sections even though those sections still exist. Or maybe the note will say, “This next section is awesome; you’ll want to read it twice.”

That’s epigenetics. Higher level cues that tell you whether or not to read a gene.

And when a scribe makes a copy of the book, they’ll not only copy all the words in the novel, but all the other stuff, too: the stuck-together pages and the margin notes.


What about my health?

Many areas of health — including cancer, autoimmune disease, mental illness and diabetes — connect with epigenetic change.

For example, scientists link epigenetic changes to neurons to depression, drug addiction and schizophrenia. And environmental toxins — such as some metals and pesticides — can cause multigenerational epigenetic effects, according to research. Once scientists and doctors decipher those processes work, they will be better equipped to treat the sick and be able to take preventative measures to help insure our health and the health of our kids.


Is it epigenetics or epigenomics?

Confusing, I know.

As we learned from the first question, epigenetics is “the study of heritable changes in gene function that do not involve changes in the DNA sequence,” according to my trusty Merriam-Webster.

Epigenomics is the study and analysis of such changes to many genes in a whole cell or organism. It’s comparable to the difference between genetics (dealing with particular pieces of DNA, usually a gene) and genomics (involving the whole genetic shebang).


Where can I learn more?

Start at this year’s Life Sciences and Society Program Symposium, “The Epigenetics Revolution: Nature, Nurture and What Lies Ahead,” on March 13-15, 2015. Speakers from all over the country will delve into the puzzles and possibilities of epigenetics.

For more background, Nature magazine also created this supplement on epigenetics.

Introducing the 11th Annual LSSP topic: The Epigenetic Revolution

Screen Shot 2015-03-05 at 3.32.13 PM

To introduce our 11th Annual Life Sciences and Society Program, The Epigenetics Revolution: Nature, Nurture and What Lies Ahead that runs at the University of Missouri March 13-15, we figured it would be nice to define the term epigenetics. Spoiler: It’s amazing and it could change everything.

According to Merriam-Webster Dictionary, epigenetics is “the study of heritable changes in gene function that do not involve changes in the DNA sequence.”


Let’s break that down. 

We can inherit something that changes what our genes do, but don’t actually change the code of our DNA.

So what sort of things do genes do?

It might be easier to think about it like this: Genes are like ingredients that make up a recipe which concocts a specific function. Each individual ingredient adds to the bigger picture. Say the recipe is our height. There are many, many genes involved in myself standing at 5 feet 6 inches and my sister towering over me at 5 feet 10 inches.

Though it’s not epigenetics that makes my sister taller than me, epigenetics could help help explain why identical twins exposed to different conditions over their lifetimes, may eventually produce offspring with extreme differences in height, as just one example.

Yesterday, Jack C. Schultz, director of the Christopher S. Bond Life Sciences Center, explained epigenetics to me this way:

“We are not simply the sum of the genes we have, but rather which ones are on or off,” Schultz said. “Those differences in gene activity explain why even identical twins are not totally identical.”

Mary Shenk, the director of this year’s LSSP symposium, said epigenetics is a revolutionary area of research that changes the way we think about genetic effects. Epigenetics research makes it clear that many aspects of the environment—including the social environment—can affect how genes are expressed, she said.

“We have always known that some traits—height, for instance—were strongly influenced by the environment through diet,” Shenk said. “But new research makes it clear just how many ‘genetic’ traits are subject to either environmental influences and/or other influences such as the sex of the parent a gene is inherited from.”

“This is a real game-changer in terms of how we see the world of genes, and makes notions of simple genetic determinism of complex traits increasingly unrealistic,” Shenk added.

The key to understanding epigenetics, is to consider the capabilities of the environment to “switch on or off” the expression of our genes.

Let’s reflect on something you may have (or haven’t) heard about: “Hogerwinter,” more well known as the Dutch Hunger Winter.  The historic famine from the winter of 1944 to the spring of 1945, has been the focal point in some of the most infamous epigenetic research.

Investigators wanted to know if prolonged famine conditions could have an effect on the offspring of pregnant mothers during that time.

“The Epigenetics Revolution: How Modern Biology Is Rewriting Our Understanding of Genetics, Disease, and Inheritance,” by Nessa Carey published by Columbia University Press in 2012, makes a compelling argument about the famine effect on gene expression in subsequent generations.

The research looked at children who were in the second trimester of their mother’s pregnancy during the winter of 1944-1945, and they found an increased incidence of schizophrenia in those children.

Carey’s research suggests epigenetics could explain effects of famine, on the expression of certain genes of the offspring of mothers pregnant during that time.


Illustration by Paige Blankenbuehler/Bond Life Sciences Center

Epigenetics are the nucleus of the 11th Annual Life Sciences and Society Program held at the University of Missouri next weekend, March 13-15. The field has the potential to unlock some of our longest standing questions about who we are and why we are this way, scientists say. The event is a great opportunity to learn more about “The Epigenetics Revolution.”

Schultz says the field of epigenetics is exploding, and it’s important to us for three big reasons.

  • One: Epigenetics helps us understand how we – or any organism – can cope with changing conditions even though we can’t change our genetic makeup.

  • Two: Epigenetics explains how traits can be passed from parent to offspring without changing genetic makeup.

  • Three: Many human diseases, including cancer, seem to involve epigenetic activity. Experiences of the parents, or of developing embryos in the womb could be responsible for difficult-to-understand problems in the offspring, such as cognitive disorders including autism spectrum disorders.

“Discovering how epigenetics works is like discovering an entirely new language,” Schultz said. “That language links our experiences – even emotional ones – to the way we are and the way our offspring look and behave.”

Schultz said exploring those links can help us understand how our environment shapes us and our societies.

According to Shenk, director of the Life Sciences and Society Program, the nine speakers coming to the MU campus all bring their own expertise to epigenetics.  As far as picking a speaker, Shenk said it’s hard to choose just one.

Nonetheless, here are a few to keep on your radar, according to Shenk:

“I am especially looking forward to hearing Annie Murphy Paul talk about her experiences writing about maternal effects for a general audience.

“Tracy Bale and Oliver Rando discuss their work on paternal effects in mice (most recent focuses on mothers instead of fathers so this is especially interesting).

“I am also excited to hear Ted Koditschek from Mizzou discuss the history of the classic Lamarckian idea of the “inheritance of acquired characteristics” and how it relates to findings from modern epigenetics,” Shenk said.

Schedule of events

The location of all speakers and affiliated events will be announced at or on the Life Sciences and Society Program — University of Missouri Facebook page.


6:30 p.m. — Topic of the talk: Sharing epigenetic research with the public. Speaker: Annie Murphy Paul, science writer and author of Origins: How the Nine Months Before Birth Shape the Rest of Our Lives. 


9 a.m. — Topic of the talk: Stress Parents: Maternal and paternal epigenetic programming of the developing brain. Speaker: Tracy Bale, professor of neuroscience and animal biology at the University of Pennsylvania.

10:30 a.m. — Topic of the talk: You are what your father ate. Speaker: Oliver Rando, professor of biochemistry and molecular pharmacology at the University of Massachusetts Medical School.

11:30 a.m. — Topic of the talk: Epigenetic inheritance and evolutionary theory: the resurgence of natural philosophy. Speaker: Massimo Pigliucci, professor of philosophy at the City University of New York.

2:15 p.m. — Topic of the talk: Environment and Autism: Past evidence, current research and future quandaries. Speaker: Irva Hetz-Picciotto, professor of public health sciences at UC Davis.

3:15 p.m. — Topic of the talk: Prenatal stress modifies the impact of phthalates on boys’ reproductive tract development. Speaker: Shanna Swan, professor of preventative medicine at Mount Sinai School of Medicine.

4:30 p.m. — Panel Session with all Saturday speakers.


9 a.m. — Topic of the talk: DOHaD, epigenetics and cancer. Speaker: Suh-mei Ho, director of the University of Cincinnati Cancer Center, and chair of the department of environmental health.

10:30 a.m. — Topic of the talk: The epigenetics of pediatric cancers. Speaker: Joya Chandra, associate professor of pediatrics at the University of Texas MD Anderson Cancer Center.

11:30 a.m. — Topic of the talk: Before epigenetics: Early ideas about the inheritance of acquired characteristics. Speaker: Ted Koditschek, professor of history at the University of Missouri.

Affiliated events: 

Exhibit running March 5-30 at the Ellis Library Collonnade. Exhibit: Generations: Reproduction, heredity and epigenetics.

1 p.m. March 9, at the Ellis Library Government Documents Section. Topic of the talk: Genes, culture and evolution. Speaker: Karthik Panchanathan, department of anthropology, University of Missouri.

3:30 o.m. March 17, at Jesse Wrench Auditorium. Topic of the talk: Profound global institutional deprivation: the example of the English and Romanian adoptee study. Speaker: Sir Michael Rutter, professor of developmental psychology at the Institute of Psychiatry at King’s College in London.

2015 Graduate Life Sciences Joint Recruitment Weekend highlights collaborative nature of research at Bond Life Sciences Center

Faculty and students crowded the hallways at Bond Life Sciences Center for an interdisciplinary poster session on Saturday. About 40 prospective graduate students listened to faculty and current graduate students from the biochemistry, interdisciplinary plant group, plant sciences, molecular pathogenesis and therapeutics (MPT), genetics area program and the life sciences fellowship program discuss their work.
The poster session was part of the 2015 Graduate Life Sciences Joint Recruitment Weekend, an event aimed at helping prospective graduate students determine if MU is the right place for them to continue their education. About 175 people participated, including current graduate students and faculty.
MU biochemistry senior Flore N’guessan said she applied to the MPT program because of her interest in virology.
“I’ve always wanted to do research,” she said.
N’guessan is currently a researcher in the Burke lab, which works on testing potential antiviral therapeutics on HIV. N’guessan has applied to other graduate programs but said that the interdisciplinary and collaborative nature of MU’s life sciences program appeals to her because it allows her to gain skills from other labs.
“It’s a collaborative and interdisciplinary university,” Dr. Jay Thelen, an associate professor of biochemistry, said. “That’s what this weekend highlights.”
Thelen emphasized that the benefit of events like the interdisciplinary poster session allows prospective students to see the diversity of science studied at Bond LSC and in labs throughout MU. And, he said, “It’s exciting to see how many students there are.”
Interested in Bond LSC? Follow us on Twitter, Instagram and Facebook.

A veterinarian abroad: Tanzania

In a second travel log from Bond LSC researcher Cheryl Rosenfeld, learn about the wildlife she encountered in Tanzania this summer. Through the North American Veterinary Community (NAVC), Rosenfeld furthered her veterinary education while encountering wildlife in their natural habitat. See more about the first leg of her trip to Rwanda here

By Cheryl Rosenfeld

In the early morning hours, our group flew from Kigali, Rwanda to the Serengeti in Tanzania. As we began the descent to the dirt runway, we glimpsed our first sight of wildebeest and the awe-inspiring Serengeti plains and I soon boarded “tano,” Swahili for the fifth 4×4 in our convoy. “Tano” soon came to have special meaning, as there are many groupings of five to see in Tanzania: “The Big Five, The Ugly Five, etc.” Emanuel, whose life-long ambition was to attend college to be a guide, led us to see all of these groups of five and then some.


Cheryl Rosenfeld

From the time we pulled away from the meeting spot, I started photographing animals on one side of the vehicle, but it seemed even more intriguing ones would appear on the other side of the road.

When the NAVC and our veterinary guide, Dr. Carol Walton, organized this trip, it was anticipated that the wildebeest would be in the Western corridor of the Serengeti but she was wrong. It’s no longer the case that the location of the wildebeest migrations can be projected with accuracy. Our lecture the first evening in Tanzania discussed how the wildebeest know to migrate and why past modeling of their migratory pattern is no longer accurate. Theories for the migratory nature of these animals include their ability to smell rain and/or changes in calcium concentrations in the soil. Wildebeest rely heavily on calcium to produce sufficient milk to nurse their calves that expend considerable energy keeping up with the herd.

In the past, the rains, like the wildebeest migration, would occur in similar sites and times throughout the year. Climate change has likely contributed to the rainfall in these sites being less reliable and correspondingly, compromised forecasting where the wildebeest will be located throughout the year. This year the wildebeest decided instead to migrate to the central Serengeti region. Thus, the next day we set off on an over-three-hour journey to this region.

While driving to find wildebeest we came upon new animals and birds including Coke’s Hartbeest, several Masai giraffe, and topis, which seemed to be splattered with oil. Finally, the wildebeest herds starting increasing in size until it reached a crescendo. As far as one could look in all directions there were wildebeest: males, females, and an abundance of baby calves. Eating alongside them were many zebras and their babies, with brown fuzzy fur along their back-end. It was the most amazing spectacle any of us had witnessed.

After seeing the vast wildebeest herds, we then came upon the elephants and lions, along with their babies. In the safety of the vehicle, we watched them engage in their natural behaviors for which no zoo experience can replicate. Both species were incredibly affectionate to the offspring and each other. In the case of the lionesses, each time the females, who were likely related, caught up with another that they had not seen in a short while would lead to emotional bouts of jumping on each other, caressing and licking. It was hardly the acts of a deadly predator. Yet, their existence rests on the wildebeest and other prey. With the wildebeest in this area, it seemed all life was flourishing at this time. A true paradise.

However, even paradise is subject to outside threats. One such threat is the massive poaching of elephants in Tanzania and surrounding African countries with 40,000 being killed last year alone. Prior to 2013, Tanzania had a “shoot to kill” policy for those caught poaching elephants or other endangered species. However, this rule has since been relaxed with the elephant numbers now in severe decline. At this unsustainable rate of poaching, the African elephant may go extinct in the wild in the next few decades.

While we did see some groups of elephants, the size of each matriarch-led group seemed less than those depicted in Sir David Attenborough and National Geographic documentaries. Moreover, one elephant that we came across, which I affectionately referred to as “Stumpy,” bore sad evidence of this brutal practice: He lost part of his trunk in a poacher’s snare. Without a full trunk, this elephant exhibited great difficulty grasping various plant items to place in his mouth.

Our subsequent travel took us to the famous “Olduvai (which should actually be Oldupai) Gorge,” one of the most famous paleoanthropological sites, including the Leakeys’ famous discovery. From there we traveled to the Ngorongoro Crater, a gigantic fracture of the earth’s crust that provides habitat for some 30,000 animals, including the most endangered black rhinoceros that only has 20 to 30 left in this area. The difference between the black and white rhino lies not in their coat color but a structural difference in their lips with black rhino possessing pointed and prehensile upper lip to browse on twigs and leaves. In contrast, white rhino possess a square upper lip to graze on the grasses below.

When Dr. Walton convinced us to set off at first light, around 6 a.m., our goal was to find one of these amazing creatures. Once again, with Emmanuel’s assistance and eagle eyes, what started out as a black dot far off in the horizon began to morph into a recognizable rhinoceros. In utter delight, we strained our necks and photographed as this beautiful animal continued to move along and forage in the distance. It would be inhumane to allow this animal to go extinct. In the case of the rhino, many Asian countries believe that its horn increases male libido, which has never been proved to be the case. This mistaken notion possibly originated from the fact that male rhinos copulate with females for several hours duration. This behavior is, however, not transferrable to men who consume any part of a rhino.

The list of species we saw in Tanzania continued to grow while we drove around the crater with black-backed and common jackals following alongside our vehicle, eland and grants gazelles grazing in the plains, flocks of greater and lesser flamingoes observed in the distance, and more lion prides and spotted hyenas.

The next day, we had our final farewell lunch at the famous Arusha Coffee Lodge, where US presidents have dined. We then set off on our long journey back to the US.

All of us were impacted by the creatures and sites we saw. One male veterinarian, who seemed gruff and quiet for most of the expedition, put it best the night before we left. He unexpectedly stood up after our last lecture and proclaimed that he signed up for the expedition more as an escape from the daily grind of being a veterinarian. However, the trip made such as impression on him to the point that he realized that part of the veterinarian oath on “relieving animal suffering” should include standing up and advocating on their behalf to prevent their brutal murder, as previously occurred in the case of the gorillas and is ongoing for elephants and rhinos. All of us were moved by his emotional comments and the tears welling up in his eyes. Our group was indeed fortunate to see these majestic animals, while they still exist, in their natural environments. I hope there is a wake-up call for nations to come together to prevent their extinction.

Trail to a Cure, Inc. helps fund training of future scientists, physcians

Over the weekend, Bond LSC HIV researchers Stefan Sarafianos, Marc Johnson and Donald Burke-Aguero joined Trail to a Cure, Inc., a Columbia nonprofit organization that helped fund important HIV research.

Since 2008, the organization has raised $74,000 for HIV/AIDS research, with some of that funding going directly to the Bond LSC providing additional hours of lab research. The 2014 online fundraising is still open and donations can be made to Trail to a Cure, Inc. until the end of the month.

The funding from Trail to a Cure helps Bond LSC researchers train future scientists and physicians in labs and in some cases, training them on the development of next generation therapies, Johnson said.


Bond LSC HIV researchers Stefan Sarafianos (left), Marc Johnson (center), and Donald Burke-Aguero (right) at the 7th annual Trail to a Cure along Katy Trail in Rocheport on May 3. | Credit: Trail to a Cure, Inc..

Bond LSC HIV researchers Stefan Sarafianos (left), Marc Johnson (center), and Donald Burke-Aguero (right) at the 7th annual Trail to a Cure along Katy Trail in Rocheport on May 3. | Credit: Trail to a Cure, Inc..

Trail to a Cure, Inc., a Columbia-based nonprofit, has raised $74,000 for HIV/AIDS since 2008. Part of the raised funds go to HIV research in the LSC, providing additional hours of lab research. | Credit: Trail to a Cure, Inc.

Trail to a Cure, Inc., a Columbia-based nonprofit, has raised $74,000 for HIV/AIDS since 2008. Part of the raised funds go to HIV research in the LSC, providing additional hours of lab research. | Credit: Trail to a Cure, Inc.

Bond LSC Investigator Chris Pires in Shanghai, Wuhan

Recently, one of our investigators, J. Chris Pires traveled to Fudan University in Shanghai and the Wuhan Vegetable Research Institute for the 19th annual Crucifer Genetic Workshop and Brassica 2014 Conference in Wuhan, China.

Pires was invited to the esteemed event as the keynote speaker of the Brassica Conference. He led workshops as part of the three-day conference March 30 through April 2 and also visited Fudan University in Shanghai during the trip.

Pires is an associate professor in biological sciences and focuses much of his research on the evolution of plants. His talk at Fudan University discussed his research on whole genome duplications and the origins of novelty in plants.

Chris Pires, investigator and associate professor in biological sciences at the Bond Life Sciences Center at Fudan University in Shanghai.

Chris Pires, investigator and associate professor in biological sciences at the Bond Life Sciences Center at Fudan University in Shanghai.


Pires with Hong Ma, the dean of life sciences at Fudan University (right) and an assistant professor (left). Pires traveled to Shanghai to lead a workshop at the university over his research in whole genome duplications in plants.


Pires at the 19th Crucifer Genetic Workshop and Brassica 2014 at the Wuhan Vegetable Research Institute in Wuhan, China. Pires was the keynote speaker at the conference.

Bond LSC staff prepares boat for April 12 fundraiser

Made completely of cardboard and Popeye themed, Bond LSC facilities crew say this boat could be the winner of the 3rd Annual Flot Your Boat for the Food Bank Race on April 12 — BLANKENBUEHLER

Made completely of cardboard and Popeye themed, Bond LSC facilities crew say this boat could be the winner of the 3rd Annual Flot Your Boat for the Food Bank Race on April 12 — BLANKENBUEHLER

Every year the College of Agriculture, Food and Natural Resources puts on a Float Your Boat for the Food Bank Race. All proceeds go to the Columbia Food Bank and last year, with 45 participants, more than $17,000 was donated.  All participants craft their own boat and obey one golden rule: cardboard only.

The Bond LSC crew are returning to the race, this year on April 12, with a Popeye themed boat they say will win it all. Cash donations are being accepted until the race day by Maureen Kemp in 106 at the Bond Life Sciences Center. The People’s Choice Award is given to the boat with the team that raised the most money for the Food Bank.

Barbie Reid, Bond LSC office support assistant, will dress as Olive Oil for this year's race. — BLANKENBUEHLER

Barbie Reid, Bond LSC office support assistant, will dress as Olive Oil for this year’s race. — BLANKENBUEHLER

The Bond LSC ATCP is a streamlined, perfectly buoyant  cardboard boat. ATCP are the letters of DNA sequencing. — BLANKENBUEHLER

The Bond LSC ATCP is a streamlined, perfectly buoyant cardboard boat. ATCP are the letters of DNA sequencing. — BLANKENBUEHLER

The Bond LSC facilities department crafted mock spinach cans to play up the Popeye theme for this year's Float Your Boat for the Food Bank Race— BLANKENBUEHLER

The Bond LSC facilities department crafted mock spinach cans to play up the Popeye theme for this year’s Float Your Boat for the Food Bank Race— BLANKENBUEHLER

Strong symposium start by Skloot

_DSC2884 2

Karin Loftin, MU Chancellor R. Bowen Loftin, Bond Life Sciences Director Jack Shultz and Tim Evans pose with Rebecca Skloot at the University of Missouri Monday evening — BLANKENBUEHLER

The bridge between public knowledge and the inner-workings of the science community is one that many are reluctant to cross. Sometimes riddled with confusing terms, the most exciting discoveries aren’t always approachable.

The 10th annual MU Life Sciences & Society Symposium began Monday evening with Rebecca Skloot as she spoke to a nearly full house at Jesse Auditorium Monday. Every year the symposium erases the line between community understanding and the discoveries of the scientific community.

Skloot, the New York Times bestselling author of The Immortal Life of Henrietta Lacks, spoke about the power of science writing in making science more approachable, gave advice to scientists on spreading the word about their discoveries and gave an insight into to the decade of reporting she did for her book. Skloot autographed copies of the book following the talk.

This year’s theme, Decoding Science, speaks to the issue of communicating scientific issues and discoveries with the general public. Skloot said scientists need to keep terms and technicalities basic and exciting.

Rebecca Skloot signs copies of her book, The Immortal Life of Henrietta Lacks, after the talk Monday at Jesse Hall

Rebecca Skloot signs copies of her book, The Immortal Life of Henrietta Lacks, after the talk Monday at Jesse Hall — BLANKENBUEHLER

Jesse Hall was filled with an eclectic mix of community, faculty and students from MU many of which lined up following the talk for nearly 30 minutes of questions.

Throughout the week, the gap between the science community and the public will be bridged with an impressive list of speakers.

The symposium, organized by the Bond Life Sciences Center which houses researchers that represent various schools at the University of Missouri, is a week-long event that features many speakers prevalent in scientific communications.

Other events to catch this week

Tuesday The “Thoughts of Plants” will be uncovered 6 p.m. at Broadway Brewery. The talk, as part of the Science Café speaker series, will be lead by Dr. Jack Shultz, director of the Bond Life Sciences Center.

Wednesday Superhero Science 11 a.m. until noon at the Colonnade in Ellis Library. Superhero submissions will be judged by the spring symposium’s own superhero: “The Antidote.” Dressed in a mask and cape, MU professor Tim Evans’ alter ego, has spicing up the field of toxicology at MU for 12 years.

Thursday James Surowiecki, a contributor to The New Yorker, will speak at 7 p.m. Thursday at Bush Auditorium, Cornell Hall. Free admission and no ticket or registration required.

Saturday All Saturday talks will be held at Jesse Hall.

10:00 a.m. Bill Nye at Jesse Auditorium, doors open at 9:00 a.m. with overflow seating available at the Monsanto Auditorium at the Bond Life Sciences Center. Tickets are sold out. Nye, most well-known for his 1990’s show Bill Nye The Science Guy, has immersed youth in “fun science” by educating in easy-to-understand terms. Nye is one of the pioneers of science communication, trying to make science more approachable by the general public.

12:30 – 1:15 p.m. Chris Mooney is a science journalist and author of Unscientific America, The Republican Brain: The Science of Why They Deny Science and Reality, and New York Times bestselling The Republican War on Science.

1:20 – 2:10 p.m. Dominique Brossard, professor and chair of the Department of Life Sciences Communication at the University of Wisconsin, Brossard studies strategic communication and public opinion in science and risk communication.

2:30 – 3:15 p.m. Liz Neeley, assistant director of Science Outreach for COMPASS, leads communications training for scientists, specializing in social media and multimedia outreach. She previously studied tropical fish evolution.

3:20 – 4:05 p.m. Barbara Kline Pope, the executive director for communications for the National Academy of Sciences, leads the Science & Entertainment Exchange, which connects top scientists with the entertainment industry for accurate science in film and TV programming.

4:10 – 5:00 p.m. A recovering marine biologist, Randy Olson is an independent filmmaker and author of Don’t Be Such a Scientist and Connection: Hollywood Storytelling Meets Critical Thinking. Olsen is a leading proponent of storytelling in science communication. His films include “Flock of Dodos” and “Sizzle,” about evolution and climate change, respectively.


Claiming Kin, MU’s 9th annual Life Sciences and Society Symposium kicks off today


Stephanie Coontz, award-winning writer on the history of marriage, chatted with Jack Schultz, director of the Bond Life Sciences Center. Coontz presented the keynote address Friday night at Claiming Kin, MU’s 9th annual Life Sciences and Society Symposium.

Check out Saturday and Sunday’s speakers on the subject of kinship at

Renowned anthropologist speaks about Noble Savages


Napolean Chagnon spoke to a full house Tuesday in Monsanto Auditorium about his new book, Noble Savages. Chagnon joined MU’s Department of Anthropology as Distinguished Research Professor and Chancellor’s Chair of Excellence in 2013. He was elected to the National Academy of Sciences in 2012, but is most known for contributions to his genealogical research, his contributions to evolutionary theory in cultural anthropology and his work in the study of warfare.

His new book is a retrospective look at his work as an anthropologist, where he most famously document the Amazonian Yanomamö tribe of Venezuela in the 1960s. His work caused controversy because it detailed a tribe violence within the tribe, an attribute at the time thought to be caused by the intrusion of modern societies on native people.

Read more about Chagnon and Noble Savages: My Life Among Two Dangerous Tribes—the Yanomamö and the Anthropologists from Mizzou Weekly.